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Dashzeveg Bayarsaihan, PhDAssociate Professor, Center for Regenerative Medicine and Skeletal Development
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Dr. Bayarsaihan received his Ph.D. from Wesleyan University and carried out postdoctoral training at Yale University. One of his research topics focuses on the molecular basis of human diseases with complex phenotype using functional genomics and animal models. Another area of interest deals with the chromatin architecture and epigenetic modifications in human and mouse neural crest stem cells. These include the global mapping studies using high resolution tiling arrays (ChIP-chip) and deep sequencing (ChIP-seq).
Not Accepting Lab Rotation Students at This Time
Journal Articles
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Single-cell RNA analysis of chromodomain-encoding genes in mesenchymal stromal cells of the mouse dental pulp.
Journal of cellular biochemistry 2024 May;
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Single-cell Transcriptome Landscape of DNA Methylome Regulators Associated with Orofacial Clefts in the Mouse Dental Pulp.
The Cleft palate-craniofacial journal : official publication of the American Cleft Palate-Craniofacial Association 2023 May;10556656231172296
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The chromatin accessibility landscape in the dental pulp of mouse molars and incisors.
Acta biochimica Polonica 2022 Feb;69(1):131-138
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Single-Cell Transcriptome Analysis Defines Expression of Kabuki Syndrome-Associated KMT2D Targets and Interacting Partners.
Stem cells international 2022 Jan;20224969441
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Single-cell transcriptome analysis defines mesenchymal stromal cells in the mouse incisor dental pulp.
Gene expression patterns : GEP 2021 Dec;119228
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Genome-wide distribution of 5hmC in the dental pulp of mouse molars and incisors.
Journal of biochemistry 2021 Oct;
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Mesenchyme-specific loss of Dot1L histone methyltransferase leads to skeletal dysplasia phenotype in mice.
Bone 2021 Jan;142115677
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Neuronal deletion of Gtf2i, associated with Williams syndrome, causes behavioral and myelin alterations rescuable by a remyelinating drug.
Nature neuroscience 2019 May;22(5):700-708
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Modus operandi of COMPASS/MLL epigenetic writers in the mammalian genome.
Epigenomics 2018 Jun;10861-863
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TFII-I and AP2α Co-Occupy the Promoters of Key Regulatory Genes Associated with Craniofacial Development.
The Cleft palate-craniofacial journal : official publication of the American Cleft Palate-Craniofacial Association 2018 Jan;55865-870
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A central role of H3K4me3 extended chromatin domains in gene regulation.
Epigenomics 2016 Jul;81011-4
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Generation of a mouse model for a conditional inactivation of Gtf2i allele.
Genesis (New York, N.Y. : 2000) 2016 May;54407-12
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Epigenetic mechanisms involved in modulation of inflammatory diseases.
Current opinion in clinical nutrition and metabolic care 2016 Apr;19263-9
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Toward integrative annotation of cell type-specific epigenomes to better understand human biology and disease.
Epigenomics 2015 Jun;7(4):519-21
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3D genome architecture in the mammalian nucleus.
Epigenomics 2013 Dec;5(6):599-601
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ChIP-Chip Identifies SEC23A, CFDP1, and NSD1 as TFII-I Target Genes in Human Neural Crest Progenitor Cells.
The Cleft palate-craniofacial journal : official publication of the American Cleft Palate-Craniofacial Association 2013 May;50(3):347-50
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What role does TFII-I have to play in epigenetic modulation during embryogenesis?
Epigenomics 2013 Feb;5(1):9-11
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Epigenetic modulation by TFII-I during embryonic stem cell differentiation.
Journal of cellular biochemistry 2012 Oct;113(10):3056-60
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PI3K/Akt-dependent functions of TFII-I transcription factors in mouse embryonic stem cells.
Journal of cellular biochemistry 2012 Apr;113(4):1122-31
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Diversity and complexity in chromatin recognition by TFII-I transcription factors in pluripotent embryonic stem cells and embryonic tissues.
PloS one 2012 Jan;7(9):e44443
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Epigenetic mechanisms in inflammation.
Journal of dental research 2011 Jan;90(1):9-17
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New TFII-I family target genes involved in embryonic development.
Biochemical and biophysical research communications 2009 Sep;386(4):554-8
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Alternative splicing and promoter use in TFII-I genes.
Gene 2009 Mar;433(1-2):16-25
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Essential functions of the Williams-Beuren syndrome-associated TFII-I genes in embryonic development.
Proceedings of the National Academy of Sciences of the United States of America 2009 Jan;106(1):181-6
Erratums
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Publisher Correction: Neuronal deletion of Gtf2i, associated with Williams syndrome, causes behavioral and myelin alterations rescuable by a remyelinating drug.
Nature neuroscience 2019 Jul;22(7):1197
Reviews
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Deciphering the Epigenetic Code in Embryonic and Dental Pulp Stem Cells.
The Yale journal of biology and medicine 2016 Dec;89(4):539-563
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Generation of neural crest progenitors from human embryonic stem cells.
Journal of experimental zoology. Part B, Molecular and developmental evolution 2010 Mar;314(2):95-103