Photo of Nilanjana  Maulik, Ph.D., FAHA

Nilanjana Maulik, Ph.D., FAHA

Professor of Surgery
Academic Office Location:
Surgery
UConn Health
263 Farmington Avenue
Farmington, CT 06030-1110
Phone: 860-679-2857
Fax: 860-679-2825
Website(s):

Cell Biology Graduate Program

Dr. Nilanjana Maulik is a Professor of Molecular Cardiology and heading the Angiogenesis Laboratory at the Department of Surgery, University of Connecticut Health Center, Farmington, Connecticut. Prof. Maulik received her Ph.D. in Biochemistry in December 1990 from Calcutta University, India. After completion of her Ph.D., Prof. Maulik joined the department of Surgery at University of Connecticut Health Center as a research fellow and continued as a faculty where now she serves as Tenured Professor. She is heavily involved in NIH funded research and serves as one of the experts in the angiogenesis field in the NIH study sections and has given more than 100 invited lectures both nationally and internationally. Prof. Maulik is also member of several prestigious societies, such as FASEB, AHA, ISHR, American College of Nutrition (ACN), International College of Angiology (ICA). She was the member of the Myocardial Ischemia Metabolism (MIM) study section of NIH for last 6 years and of the NHLBI Program Project Review Committee. Presently, Prof. Maulik serves as a reviewer of a several special emphasis panel study section of NIH. She is also a reviewer of the AHA Grant review process. She is on several Editorial boards of major cardiovascular journals. She is an associate editor of Molecular Cellular Biochemistry journal. Teaching is an integral part of her professional path. She is a recipient of several prestigious awards including Faculty Recognition award from the University of Connecticut Health Center. Her research focuses on the molecular mechanism of myocardial angiogenesis in the diseased/infarcted heart, ischemia/reperfusion injury, apoptosis and on the development of cardioprotective strategies which includes gene and stem cell therapy. She has published 170 original peer reviewed articles and 35 book chapters and very recently (2010) she has edited a book on epigenetics and human diseases for CRC press. Prof. Maulik has organized several international conferences/symposia, etc.

Education
DegreeInstitutionMajor
B.Sc.St. Xavier's CollegeChemistry (Hons)
M.Sc.University of CalcuttaBiochemistry
Ph.D.University of CalcuttaBiochemistry
Ph.D.University of Connecticut School of MedicineMolecular Cardiology

Awards
Name of Award/HonorAwarding Organization
Fellow of American College of NutritionAmerican College of Nutrition
Faculty Recognition AwardUniversity of Connecticut Health
Fellow of American Heart AssociationAmerican Heart Association
Best Oral Presentation Randox Award at International Society for Free Radical Research
Grant-In-AidAmerican Heart Association
Young InvestigatorAmerican College of Angiology
Post Doctoral FellowshipAmerican Heart Association
Name & DescriptionCategoryRoleTypeScopeStart YearEnd Year
Scholarship and Promotion Committee, Department of SurgeryAdvisory CommitteeMemberUConn HealthUniversity20192024
Medical Curriculum Evaluation Committee (CEC)Advisory CommitteeMemberUConn HealthUniversity20182023
Group of Women in Medicine and Science (GWIMS)Advisory CommitteeMemberUConn HealthUniversity20172023
Experimental Biology and MedicineProfessional/Scientific OrganizationAdhoc ReviewerExternalInternational20152021
Expert Opinion on Biological TherapyProfessional/Scientific JournalAdhoc ReviewerExternalInternational2015
NanomedicineProfessional/Scientific JournalAdhoc ReviewerInternational20152019
Editor-In-Chief for the special Issue "Antioxidants in Longevity and Medicine" in the Journal Oxidative Medicine and LongevityEditorial BoardEditor-In-ChiefExternalInternational20142014
Molecular Biology ReportsEditorial BoardEditor-In-ChiefExternalInternational20132020
Editor-In-Chief for the special Issue "Antioxidants in Longevity and Medicine" in the Journal Oxidative Medicine and LongevityProfessional/Scientific JournalEditor-In-ChiefExternalInternational20132013
International Conference on Natural Products in Health and 25th Annual Conference of The Indian Society for Atherosclerosis Research, Annamalai University, Annamalainagar, Tamilnadu, IndiaProfessional/Scientific OrganizationOrganizerExternalInternational2012
International Academy of Cardiology Annual Scientific SessionAdvisory CommitteeAdvisorExternalInternational20122023
International Society for Free Radicals (SFRR), Chennai, India Professional/Scientific OrganizationOrganizerExternalInternational2011
PLoS ONEOtherExternalInternational20102020
International Academy of Cardiovascular Sciences, University of Mannitoba, St. Boniface General Hospital Research Center, Canada.OtherInternational20082018
CirculationOtherExternalInternational20042018
Graduate Program Committee (Vascular and Cell Biology)Education CommitteeFacultyUConn HealthUniversity20042020
Society for Free Radical Research (SFRR)Professional/Scientific OrganizationAdvisorExternalInternational20042020
American Heart AssociationProfessional/Scientific OrganizationFellow of American HeartExternalInternational20042022
Journal of Cellular Molcular MedicineOtherInternational20032019
NIH Study SEctionStudy SectionReviewerExternalNational20032020
The American Physiological SocietyProfessional/Scientific OrganizationMemberInternational20022020
Molecular Cellular BiochemistryEditorial BoardReviewerExternalInternational20012020
Antioxidant Redox SignalingEditorial BoardMemberInternational20002020
DiabetesOtherInternational20002020
Antioxidant Redox SignalingEditorial BoardMemberInternational20002024
American Journal of PhysiologyProfessional/Scientific JournalAdhoc ReviewerExternalNational20002020
American Heart AssociationProfessional/Scientific OrganizationPremium Professional Silver Heart MemberExternalInternational19982020

Inspite of significant improvements in its medical and surgical management Ischemic Heart Disease (IHD) is still the leading cause of morbidity and mortality in the western world. The health care system of the modern world has come up with good therapeutic methods, in time, to fight the devastating effects of the disease but it doesn’t seem to be enough to tackle still many aspects of the disease. Now the pressure is even greater on basic scientists and clinicians to deliver their best to translate their knowledge from bench to bedside.


Dr Maulik and her team is dedicated to bring about better understanding of the molecular mechanisms that govern the pathology of IHDs and the processes of angiogenesis and arteriogenesis in cellular and animal models of heart disease and to apply this basic knowledge obtained to tackle this dreaded disease by employing newer and better therapeutic approaches to protect, rescue, repair or regenerate the myocardium from ischemia induced myocardial degeneration and failure.


The process of cellular regeneration and repair which leads to reconstruction of structural and functional normal tissue in an injured area is undoubtedly highly efficient in embryos. However this ability is lost at the time of birth. During the fetal life both tissue repair refer to an identical biological response to injury, however, after birth they refer to two distinct biological processes. In the case of tissue repair, an injury results in repair and scar formation in all organs while on the other hand regeneration implies the replacement of dead cells by newly formed cells that differentiate and organize aiding in the restoration of the original structure of the lost tissue. This process of tissue regeneration occurs during the normal physiological cell turnover process, however in the absence of injury.


Several attempts have been made to promote cardiac regeneration in the infarcted ventricle. However despite many efforts to unearth a suitable strategy, the most appropriate form of myocardial regeneration therapy after myocardial injury remains to be identified. Over the past few years the better understanding of the biology of vasculogenesis, angiogenesis and arteriogenesis has significantly increased the use of growth factors in the treatment and/or prevention ischemic heart disease by inducing blood vessel formation and thereby restoring/enriching the blood flow. Therapeutic angiogenesis makes use of the administration of a drug or angiogenic growth factor protein or gene to promote the development of endogenous collateral vessels in ischemic myocardium. Gene transfer and protein therapy are the two main approaches that have been used to achieve therapeutic angiogenesis. Most recently, interest has grown in the potential of cell-based gene transfer to induce myocardial angiogenesis and arteriogenesis. It has been established in many preclinical studies that bone marrow cells may be able to secrete multiple potentially angiogenic substances as well as differentiate into cells that create new blood vessels. But the concept of myogenesis is not far behind as it is known that there may be dedicated precursors of cardiomyocytes in the myocardium, however, it seems to be little bit more far fetched in relation to the differentiation of bone-marrow-derived cells into heart muscle cells. It still remains elusive whether the transplanted cells themselves differentiate, or whether the paracrine effects from these transplanted cells stimulate already resident stem cells in the heart to differentiate.


Dr. Maulik’s research strategy combines expertise in different model systems to study IHD and the processes of angiogenesis and arteriogenesis, in vitro (cell culture), ex vivo (Langendorff and working heart model of myocardial global ischemia) and in vivo (rat and mouse survival surgery/LAD occlusion model myocardial regional ischemia) along with the various molecular biology techniques, proteomics, genomics, immunohsitochemical technique, small animal echocardiographic analysis and other imaging techniques that are made available to the fellows. Her laboratory also have expertise in non-invasive administration of drugs, adenovirus associated gene transfer and cell-based gene transfer to the myocardium to study the effect of inducing or turning off genes of interest in the process or angiogenesis, arteriogenesis and myocardial regeneration.


Her laboratory currently focuses on:


1) Molecular and cellular signaling during myocardial ischemia and reperfusion.
2) Mechanism of pharmacological as well as ischemic preconditioning of the heart.
3) Pharmacological preconditioning of sick hearts as in the case diabetes, hypertension, hypercholesterolemia etc.
4) The role of ubiquitination and proteasomal degradation of different proteins in sick hearts.
5) Impairment of myocardial angiogenesis in diabetes: How to cope and Thrive
6) Adenovirus associated gene transfer and cell-based gene transfer to study the effect of inducing or turning off genes of interest in the process of myocardial regeneration.
7) Molecular mechanism of angiogenesis and arteriogenesis in hind limb ischemia model.


Science is quickening its pace towards better ideas in every field it has stepped into, particularly in the field of medicine. A time may not be far when we might be able to effectively cure IHD. But, “prevention is always better than cure”.

Not accepting students for Lab Rotations at this time

Journal Articles

Books

  • N Maulik Cardiovascular Diseases: Nutritional and Therapeutic Interventions 2012 Jan;
  • N Maulik Nutrition, epigenetic mechanism, and human disease 2010 Jan;

Book Chapters

  • Resveratrol Emerges As a Miracle Cardioprotective Phytochemical Polyphenol and Nutraceutical, Chapter 21
    Selvaraju S, Joshi M, Kotha SR, Parinandi NL, Maulik N. Cardiovascular Diseases: Nutritional and Therapeutic Interventions 2012 Jan;401-420
  • The effects of resveratrol on Diabetes and obesity, Chapter 33
    Sanchez JA, Thirunavukkarasu M and Maulik N. Nutritional and Therapeutic Intervention of Diabetes and Metabolic Syndrome 2011 Jan;413-430
  • Aging by Epigenetics: Nutrition, An Epigenetic key to Long Life, Chapter 2
    Maulik N and Maulik G. Nutrition, Epigenetic Mechanisms, and Human Disease 2010 Jan;13-30

Abstracts

  • Pellino-1 overexpression in cardiomyocyte enhances cardiac function and attenuates ventricular remodeling in murine myocardial infarction model.
    Jacob Campbell, Vaithinathan Selvaraju, Mahesh Thirunavukkarasu,Coca-Soliz Vladamir, Leonidias Tapias, Kurapaty Venkatapoorna Chandramohan, Siu-Pok Yee, David W McFadden, J.Alexander Palesty, Maulik N. Journal of the American College of Surgeons 2017 Oct;225S33
  • Pellino1 is required to preserve myocardial function and promote neovascularization in the infarcted myocardium
    Thirunavukkarasu M, Oriowo B, Selvaraju V, Joshi M, Tapias L, Coca-Soliz V, Saad I, Sanchez JA, Palestey JA, Yee S-P and Maulik N. Circulation 2016 Nov;A15101-A15101
  • Adeno-hspa12b delivery improves neovascularization in a murine model of hind limb ischemia
    Rishi MT, Selvaraju V, Palestey JA, Maulik N. Circulation 2016 Nov;134A15180-A15180
  • Regeneration of infarcted myocardium by genetically modified stem cells
    Nilanjana Maulik, Sumanth C Suresh, Vaithinathan Selvaraju, Mahesh Thirunavukkarasu, Aaftab Hussain, Juan A Sanchez and J Alexander Palesty J Stem Cell Res Ther 2016 Aug;
  • Deletion of newly described molecule, pellino1, aggravates blood perfusion and neovascularization and exerts increased tissue fibrosis in a murine model of hindlimb ischemia
    Rishi MT, Shaikh IA, Selvaraju V, McFadden DW, Palesty JA, Maulik N Journal of the American College of Surgeons 2015 Oct;221:S178
  • Pellino1, a novel molecule in the vascular endothelial growth factor (vegf) signaling pathway, rescues left ventricular function and angiogenesis after myocardial infarction in flk-1+/-mice
    Saad I, Mahesh T, Coca-Soliz V, Selvaraju V, Palesty JA, Maulik N. Journal of the American College of Surgeons 2015 Oct;S221:S25
  • Severe necrosis of ischemic skin flap tissue in flk-1+/- (vascular endothelial growth factor r2/kdr) knockout mice treated with pellino 1(peli1) gene therapy
    Wilson RL, Rednam CK, Selvaraju V, Rishi MT, Mahesh T, Palesty JA, McFadden DW, Maulik N. Journal of the American College of Surgeons 2015 Oct;221:S119
  • Enhanced akap12/gravin expression leads to myocardial injury by disrupting angiogenic signals related to hsp/thioredoxin-1/vegf cascade in type i diabetic rats
    Vaithinathan Selvaraju, Sumanth C Suresh, Mahesh Thirunavukkarasu, J.Alexander Palesty, Juan A Sanchez, Nilanjana Maulik Circulation 2014 Nov;130A15397-A15397
  • Intramyocardial gene therapy with a novel angiogenic molecule, pellino-1 preserves functions and increases cell survivability in a mouse myocardial infarction model
    Coca-Soliz V, Tapias L, Selvaraju V, Thirunavukkarasu M, Palesty JA, Maulik N. Circulation 2014 Nov;
  • Thereapeutic angiogenesis induced by human Trx-1 gene in mice hind limb ischemia model: Preclinical study for treatment of peripheral artery disease.
    Mahesh Thirunavukkarasu Inam A Shaikh, Vaithinathan Selvaraju, J.Alexandar Palesty, Nilanjana Maulik Circulation 2014 Nov;130A17955-A17955
  • Deletion of flk-1 and its target protein,mapkinase-2 impairs neovascularization and perfusion in a murine hindlimb ischemia model: A double knockout study.
    Abunnaja SS, Selvaraju V, Thirunavukkarasu M, Rahman L, Sanchez JA, McFadden DW, Palesty JA, Maulik N. Journal of the American College of Surgeons. 2014 Oct;S219:S161
  • Effects of prolyl-4-hydroxylase 2 (phd-2) gene deletion on cardiac microrna expression and ventricular function after myocardial infarction: A cardiac-specific mouse knockout model
    Shaikh IA, Joshi M, Selvaraju V, Thirunavukkarasu M, Tapias L, Sanchez JA,Palesty JA, McFadden DW, Maulik N Journal of the American College of Surgeons 2014 Oct;3S29.
  • Overexpression of glutaredoxin-1 stimulates revascularization and improves blood perfusion in a murine hind-limb ischemia model
    Abunnaja S, Selvaraju V, Thirunavukka M, Mcfadden D, Palesty JA, Maulik N. Journal of the American College of Surgeons 2013 Oct;217S28
  • Thioredoxin-1 (trx-1) engineered mesenchymal stem cell therapy increased proangiogenic factors, reduced fibrosis and improved heart function in the infarcted rat myocardium
    Thirunavukkarasu M, Suresh SC, Selvaraju V, Sanchez JA, Palesty JA, McFadden DW, Maulik N Journal of the American College of Surgeons. 2013 Oct;3S34
  • Myocardial gene therapy by adeno-hspa12b administration induces angiogenesis and prevents ventricular dysfunction following acute myocardial infarction in type i diabetic rats
    Thirunavukkarasu M, Suresh SC, Selvaraju V, Sanchez JA, Maulik N. Circulation 2012 Nov;126A18778
  • Prolyl hydroxylase 1 silencing promotes angiogenesis and improves blood perfusion in a mouse hind-limb ischemia model: A gene knockout study
    Rishi MT, Thirunavukkarasu M, Selvaraju V, Sanchez JA, Maulik N Circulation 2012 Nov;126A14398
  • Vegfr2 (flk-1+/-) gene knockout leads to reduced angiogenesis, ventricular dysfunction, and alterations in micro-rna profile following myocardial infarction
    Joshi M, Thirunavukkarasu M, Selvaraju V, Sanchez JA, Maulik N. Circulation 2012 Nov;126A14914
  • Intramyocardial gene therapy with adeno beta-catenin preserves cardiac function by increased angiogenesis and cell survival in type I diabetic rat.
    Suresh SC, Thirunavukkarasu M, Selvaraju V, Maulik N Journal of the American College of Surgeons 2012 Oct;215S33
  • Silencing prolyl hydroxylase-3 (phd-3) improves blood perfusion in a murine hind-limb ischemia model
    Rishi MT, Thirunavukkarasu M, Selvaraju V, Sanchez JA, Maulik N Journal of the American College of Surgeons 2012 Oct;215S151
  • Statin therapy in diabetic rats attenuates myocardial dysfunction via a skp2/p27 pathway after myocardial infarction.
    Joshi M, Thirunavukkarasu M, Selvaraju V, Sanchez JA, Maulik N. American College of Surgeons 2012 Oct;
  • Sequencial Activation of VEGF/Flk-1/MKK2/NFkappaB Signaling in Ischemic Preconditioning Induced Neovascularization: A Study with Flk-1+/– and MKK2–/– Knockout Mice Circulation
    Mahesh Thirunavukkarasu; Yozu Akita; Samson Mathews Samuel; Lijun Zhan; Chi-Kuang Huang; Matthias Gaestel; Nilanjana Maulik 2008 Jan;118S_575-S_576
  • Pellino-1: A Novel Candidate In Vegf/Flk-1/MKK2 Signaling In Ischemic Preconditioning Induced Cardioprotection: A Study Using Flk +/– and MKK2 –/– Knockout Mice
    Mahesh Thirunavukkarasu ,Samson Mathews Samuel Sankar Addya; Lijun Zhan; Chi-Kuang Huang; Matthias Gaestel; Venogopal P Menon; Nilanjana Maulik Circulation. 2007 Jan;116II_213
  • Mitogen-activated Protein Kinase-activated Protein Kinase 2 (MK2) Deficiency Protects Heart from Ischemia/Reperfusion Injury in Mice
    Shigeaki Kaga, Lijun Zhan, Chi-Kuang Huang, Matthias Gaestel, and Nilanjana Maulik AHA annual meeting 2005 Jan;
  • Prolyl Hydroxylase 1 Silencing Promotes Angiogenesis and Improves Blood Perfusion in a Mouse Hind Limb Ischemia Model: A Gene Knockout Study
    Rishi, M.T., Thirunavukkarasu, M., Selvaraju, V., Sanchez, J.A., Maulik, N. Circulation 12614398
  • Silencing Prolyl Hydroxylase Domain Protein 3 Stabilizes Hypoxia Inducible Factor −1 Alpha and Preserves Myocardial Function Through the Activation of Proangiogenic and Anti-Apoptotic Pathway Following Myocardial Injury in Mice
    Oriowo, B., Thirunavukkarasu, M., Adluri, R.S., Zhan, L., Takeda, K., Fong, G.H., Sanchez, J.A., Maulik, N. Journal of the American College of Surgeons 213(3):S33-S34

Editorials

Letters

Other

  • Journal selected Cover page from our study
    Shaikh IA, Rishi MT, Youssef M, Selvaraju V, Thirunavukkarasu M, Ukani G, Lakshmanan R, Palesty JA, Maulik N. The Journal of surgical research 2017 Oct;216

Reviews

Short Surveys

Title or AbstractTypeSponsor/EventDate/YearLocation
Angiogenic Response in Ischemic TissueLectureSociety of Free radical Research2018Delhi, India
Therapeutic Angiogenesis in Ischemic HeartTalkGangaram Hospital2018Delhi, India
VEGF in Wound RepairTalkJadavpur University2018Kolkata, India
Regulation of Angiogenic Response in Diabetic MyocardiumPlenary LectureWorld Congress Cardiology2018Boston
CARDIOVASCULAR PROTECTION, REPAIR AND REGENERATIONOtherWorld Congress Cardiology2018Boston
Bioengineered Nanofiber Scaffold Loaded with Resveratrol Improves Cardiac Function Following Myocardial Infarction After Four WeeksTalk25th Annual NESS Presentation Day2018Waltham Woods in Waltham, Massachusetts
Utilization of a Novel Engineered Nanofibrous Scaffold Loaded with Resveratrol in a Preclinical Full Thickness Wound Model in MiceTalk25th Annual NESS2018Waltham Woods in Waltham, Massachusetts
New Molecular Targets of VEGF Signaling in Cardiovascular Disease.LectureIACS, North America2017Orlando, Florida
Proangiogenic Molecules in Cardiac Disease:LectureInternational Society of Heart Research Conference2017Chennai, India
Cardiac Disease and its TherapyOtherIACS, North America2017Orlando, Florida
Group Lecture on "VEGF Signaling in Cardiovascular System".OtherUniversity of Calcutta2017Calcutta, India
Bioengineered Nanofiber Scaffold Loaded with Resveratrol Improves Cardiac Function Following Myocardial Infarction: Our Preliminary Findings.TalkConnecticut Chapter of the American College of Surgeons2017Connecticut
Pellino-1 Overexpression in Cardiomyoctes Enhances Cardiac Function and Attenuates Ventricular Remodeling in Murine Myocardial Infarction Model.TalkAmerican College of Surgeons2017San Diego, CA
Cardiac Regeneration and RepairOtherInternational Academy of Cardiovascular Sciences2016Sherbrooke, Quebec, Canada
New Molecular Targets of VEGF Signaling in Cardiovascular Disease,LectureInternational Academy of Cardiovascular Sciences2016Sherbrooke, Quebec, Canada
Regeneration of Infarcted myocardium by genetically modified stem cells.Plenary LectureStem cell congress2016Manchester, UK
Stem Cell in tissue repair and regenerationOtherStem Cell Congress2016Manchester, UK
Thioredoxin-1 Overexpression in mice decreases sepsis-induced cardiac failureTalkSociety of Black Academic Surgeons2016Columbus, Ohio
Adeno-HSPA12B Delivery Improves Neovascularization in a Murine Model of Hind Limb IschemiaPosterAmerican Heart Scientific Session2016New Orleans
Pellino1 is Required to Preserve Myocardial Function and Promote Neovascularization in the Infarcted MyocardiumPosterAmerican Heart Scientific Session2016New Orleans
Thioredoxin-1 Augments Wound Healing and Promotes Angiogenesis in a Murine Ischemic Full Thickness Wound Model.TalkPlastic Surgery The Meeting2016Los Angeles, California
Phyllanthus emblica in cardioprotection. 8th International Symposium of Ayurveda and Health. Health, Immunity &Inflammation: Integrating Ancient Approaches with Modern Concepts.TalkUniversity of Connecticut2016Farmington, CT
Various In Vivo Ischemic MOdels to Study Therapeutic AngiogenesisLectureUniversity of Connecticut Health, Surgery2015Department of Surgery
Adeno-Thioredoxin- 1 Delivery Improves Neovascularization in a Murine Model of Hind Limb Ischemia.TalkSurgical Congress2015Las Vegas
Improving Left Ventricular Function and Angiogenesis after MI in Flk Knock Out Mice with Intramyocardial Gene Therapy with Pellino1, a Novel Molecule in VEGF Signaling PathwayTalkNew England Surgical Society2015
Pellino 1 Gene Therapy Shows Novel Proangiogenic Function by Improving Survivability of Ischemic Skin Flap Tissue in Flk-1 Knockout Mice via NO/HO-1 Signaling Cascade.TalkNew England Surgical Society2015
Severe Necrosis of Ischemic Skin Flap Tissue in Flk-1+/- (Vascular Endothelial Growth Factor R2/KDR) Knockout Mice Treated with Pellino 1 (Peli1) Gene Therapy.TalkThe American College of Surgeons2015Chicago, IL
Key players in cardiac and peripheral artery disease: potentials and challengesTalkInstitute of Cellular and System Medicine, National Health Research Inst.2015Zhunan, Taiwan
Silencing of prolylhydroxylases in cardiac and peripheral artery disease: potentials and challengesPlenary LectureNational Cheng Kung University, Taiwan2015Tainan, Taiwan
Key players in Cardiac Disease: Potential and ChallengesLecture11th International Symposium on Pharmaceutical Sciences, Ankara, Turke2015Ankara University, Turkey
Key Players in Myocardial Angiogenesis: Potential and ChallengesLectureInternationl Academy of Cardiovascular Sciences: North American Section2015Creighton University, Omaha, Nebraska
New Molecular Targets of VEGF Signaling in Cardiovascular DiseaseLectureInstitute for heart Research, Slovak Academy of Scences2015Bratislava, Slovak Republic
Silencing of Prolylhydroxylases in Cardiac Repair: Potential and ChallengesPlenary LectureInternational Academy of Cardiology: 19th World Congress on Heart Disease,2014Boston, MA
Key Players in Cardiac & Peripheral Artery Disease: Potential and ChallengesLecture2nd Cardiovascular Forum for Promoting Centers of Excellence and Young Inve2014Winnipeg, Manitoba, Canada
Key Players in Cardiac & Peripheral Artery Disease: Potential and ChallengesLecture41st National Conference of Association of Clinical Biochemists of India2014Jodhpur, India
SILENCING OF PROLYLHYDROXYLASES IN CARDIAC REPAIR: POTENTIAL AND CHALLENGESOther2013East Virginia
SILENCING OF PROLYLHYDROXYLASES IN CARDIAC REPAIR: POTENTIAL AND CHALLENGESPlenary LectureClinical Conference2013Kalyani, India
SILENCING OF PROLYLHYDROXYLASES IN CARDIAC REPAIR: POTENTIAL AND CHALLENGESLectureThe Cardiovascular Forum for Promoting Centers of Excellence and Young Inve2013Louisville, KY
SymposiumOtherClinical Conference2013Kalyani, India
SymposiumOtherThe Cardiovascular Forum for Promoting Centers of Excellence and Young Inve2013Louisville, KY
THIOREDOXIN-1 GENE THERAPY FOR CARDIOVASCULAR DISEASELectureInternational Academy of Cardiology: 18th World Congress on Heart Disease,2013Vancouver, B.C., Canada
Inhibition of Prolyl Hydroxylases: a Novel therapy for Peripheral Arterial and Cardiovascular DiseasesPlenary Lecture25th Annual Conference of The Indian Society for Atherosclerosis Research2012Annamalai nagar, Tamilnadu, India
Silencing of Prolylhydroxylases in Cardiac and Peripheral artery disease: Potential and ChallengesPlenary LectureInternational Meet on Advanced Studies in Cell Signaling Network (CESIN 2012012Kolkata, India
Diabetes-mediated impairment of angiogenesis in myocardial injuryLecture53rd Annual American College of Nutrition2012Morristown, NJ
Silencing of prolyl hydroxylases in cardiac Repair: Potential and challengesLectureInternational Conference on Recent Trends in Therapeutic advancement of Fre2011Chennai, India
Silencing of prolyl hydroxylases in cardiac Repair: Potential and challenges. Lecture4th World Congress of international Academy of Cardiovascular Sciences2011Vadodara, India
Resveratrol expanding roles in cardiovascular disordersLecture8th International Conference of Functional Foods for the Prevention and management of chronic Diseas2011Las Vegas, Nevada
Rescue of diabetes related impairment of myocardial angiogenesis: Potential and challengesLectureUniversity Gill Heart Institute and Cardiovascular Research Center’s Cardiovascular Seminar Series f2011Lexington, KY
Silencing of prolyl hydroxylases in cardiac Repair: Potential and challengesLectureWinnipeg Heart International Conference2011Winnipeg, Canada
Thereapeutic Angiogenesis: Treating the coronary Vessel disease with genes and Gene productsLecture97th Indian Science Congress2010Trivandrum, Kerala, India
Rejuvenating the failing heart: Messenchymal stem cells making headway towards clinicsLectureSociety for Free Radical Research-India Satellite Conference2010Thrissur, Kerala, India
Rescue of diabetes related impairment of myocardial angiogenesis: Potential and challengesLectureInternational Conference on Advances in Free Radical Research, Natural products, Antioxidants and Ra2010Hydrabad, India
Rescue of diabetes related impairment of myocardial angiogenesis: Potential and challengesLectureOCC 2010 World Congress on Translational Redox and Tissue Oxygen Biology2010Santa Barbara, California